Figure 4
Figure 4. Expression analysis of human FMN2 gene in different types of B leukemic samples. Semiquantitative RT-PCR analysis was performed on 12 pediatric pre-B-ALL (lanes 1-12), 7 adult pre-B-ALL (lanes 13-19), 2 Burkitt leukemias (lanes 20 and 21), one mantle cell lymphoma (lane 22), one follicular lymphoma (lane 23), one B-cell prolymphocytic leukemia (lane 24), and one chronic lymphocytic leukemia (lane 25). RT-PCRs were performed in triplicate for each gene. The actin gene was used as an internal control as described in “Semiquantitative RT-PCR,” and expression level in each leukemia is presented as a selected gene/actin density ratio. Statistical analysis was performed using one-way analysis of variance, and P less than .05 was considered to be significant (*P < .05, **P < .01, ***P < .001) compared with the respective control (CH).

Expression analysis of human FMN2 gene in different types of B leukemic samples. Semiquantitative RT-PCR analysis was performed on 12 pediatric pre-B-ALL (lanes 1-12), 7 adult pre-B-ALL (lanes 13-19), 2 Burkitt leukemias (lanes 20 and 21), one mantle cell lymphoma (lane 22), one follicular lymphoma (lane 23), one B-cell prolymphocytic leukemia (lane 24), and one chronic lymphocytic leukemia (lane 25). RT-PCRs were performed in triplicate for each gene. The actin gene was used as an internal control as described in “Semiquantitative RT-PCR,” and expression level in each leukemia is presented as a selected gene/actin density ratio. Statistical analysis was performed using one-way analysis of variance, and P less than .05 was considered to be significant (*P < .05, **P < .01, ***P < .001) compared with the respective control (CH).

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