Figure 1
Figure 1. SHIP-1 Fli-1 protein expression are inversely correlated in mouse erythroleukemia cells. SHIP-1, Fli-1 (A) and PTEN (B) expression levels in HB1.1, CB3, HB9.2, and HB22.2 cells, derived from spleens of F-MuLV–infected Balb/c mice, and HB60-5, DP16-1, and DP17-17 cells, derived from spleens of FV-P (SFFV-P + F-MuLV)-infected DBA/2J adult mice. SHIP-1 and Fli-1 expression levels in (C) the spleens of 8-week-old F-MuLV–infected mice (B2-B4). (D) Exogenous expression of Fli-1 in DP17-17 cells (MigR1 Fli-1) results in decreased expression of SHIP-1 compared with nontransduced (N/T) and empty vector control (MigR1) infected cells. β-Actin was used as a loading control for all samples.

SHIP-1 Fli-1 protein expression are inversely correlated in mouse erythroleukemia cells. SHIP-1, Fli-1 (A) and PTEN (B) expression levels in HB1.1, CB3, HB9.2, and HB22.2 cells, derived from spleens of F-MuLV–infected Balb/c mice, and HB60-5, DP16-1, and DP17-17 cells, derived from spleens of FV-P (SFFV-P + F-MuLV)-infected DBA/2J adult mice. SHIP-1 and Fli-1 expression levels in (C) the spleens of 8-week-old F-MuLV–infected mice (B2-B4). (D) Exogenous expression of Fli-1 in DP17-17 cells (MigR1 Fli-1) results in decreased expression of SHIP-1 compared with nontransduced (N/T) and empty vector control (MigR1) infected cells. β-Actin was used as a loading control for all samples.

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