Figure 1
Figure 1. Association between KIR3DS1 genotype and PFS. (A) PFS for GR patients who were KIR3DS1+ was significantly shorter than for GR patients who were KIR3DS1− (P = .003). The median PFS for poor-risk patients was not significantly different from GR, KIR3DS1+ patients (P = .061). (B) Among patients with GR disease at ASCT, those with KIR3DS1+/KIR3DL1+/HLA-Bw4− have significantly reduced PFS than patients with KIR3DS1+/KIR3DL1+/HLA-Bw4+ and patients with KIR3DS1− irrespective of HLA-Bw4 (P = .002).

Association between KIR3DS1 genotype and PFS. (A) PFS for GR patients who were KIR3DS1+ was significantly shorter than for GR patients who were KIR3DS1 (P = .003). The median PFS for poor-risk patients was not significantly different from GR, KIR3DS1+ patients (P = .061). (B) Among patients with GR disease at ASCT, those with KIR3DS1+/KIR3DL1+/HLA-Bw4 have significantly reduced PFS than patients with KIR3DS1+/KIR3DL1+/HLA-Bw4+ and patients with KIR3DS1 irrespective of HLA-Bw4 (P = .002).

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