Proposed model of ECSCR serving as a molecular switch. In the presence of ECSCR, VEGF induces a rac-reactive oxygen-notch chemotactic pathway. In the absence of ECSCR, alternative survival pathways are activated (rho, phosholipase D).
Proposed model of ECSCR serving as a molecular switch. In the presence of ECSCR, VEGF induces a rac-reactive oxygen-notch chemotactic pathway. In the absence of ECSCR, alternative survival pathways are activated (rho, phosholipase D).
