Figure 5
Figure 5. ΔNp63 dysregulation contributes to the rps19-deficient phenotype. Nonneural ectoderm () is expanded in rps19-deficient embryos at the expense of neural one (◀). In situ hybridization with p63 and gata2 probes. Hybridization with gsc probe (red arrow) was used to mark the dorsal side. Tilted dorsal view. Thirty wild-type or morphant embryos were used in the reaction; 7 or 8 from the morphant group had visible expansion of the nonneural ectoderm markers. Representative embryos are shown. Schema of zebrafish fate map at 6 hpf shows side view.

ΔNp63 dysregulation contributes to the rps19-deficient phenotype. Nonneural ectoderm () is expanded in rps19-deficient embryos at the expense of neural one (◀). In situ hybridization with p63 and gata2 probes. Hybridization with gsc probe (red arrow) was used to mark the dorsal side. Tilted dorsal view. Thirty wild-type or morphant embryos were used in the reaction; 7 or 8 from the morphant group had visible expansion of the nonneural ectoderm markers. Representative embryos are shown. Schema of zebrafish fate map at 6 hpf shows side view.

Close Modal

or Create an Account

Close Modal
Close Modal