Figure 3.
Figure 3. Effect of phagocytosis on actin polymerization and protein tyrosine phosphorylation in septic neutrophils. (A) Phagocytic index of neutrophils from controls (□) or patients with sepsis (▦), treated (+) or not (–) with CyB (15 μg/mL) or with genistein (Gen; 80 μM). Data are means ± SD. *P < .05 compared with controls (ANOVA followed by Bonferroni). Fluorescence microscopy (magnification: × 1000) for F-actin (B) and phoshotyrosine (C) of neutrophils from controls (i; □) and patients with sepsis (ii; ▦), after phagocytosis of opsonized zymosan, performed as described in “Patients, materials, and methods.”

Effect of phagocytosis on actin polymerization and protein tyrosine phosphorylation in septic neutrophils. (A) Phagocytic index of neutrophils from controls (□) or patients with sepsis (▦), treated (+) or not (–) with CyB (15 μg/mL) or with genistein (Gen; 80 μM). Data are means ± SD. *P < .05 compared with controls (ANOVA followed by Bonferroni). Fluorescence microscopy (magnification: × 1000) for F-actin (B) and phoshotyrosine (C) of neutrophils from controls (i; □) and patients with sepsis (ii; ▦), after phagocytosis of opsonized zymosan, performed as described in “Patients, materials, and methods.”

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