Fig. 4.
Fig. 4. Sialidase treatment and compound ST3Gal deficiencies implicate multiple sialyltransferases in the formation of selectin ligands in vivo. / E- and P-selectin–immunoglobulin chimera binding was assessed following treatment of peripheral blood Gr1+ cells from ST3Gal-IV–deficient mice with Arthrobacter ureafaciensneuraminidase.

Sialidase treatment and compound ST3Gal deficiencies implicate multiple sialyltransferases in the formation of selectin ligands in vivo.

E- and P-selectin–immunoglobulin chimera binding was assessed following treatment of peripheral blood Gr1+ cells from ST3Gal-IV–deficient mice with Arthrobacter ureafaciensneuraminidase.

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