Figure 1
Hypothesis on the role of PAX5 fusion proteins to sustain survival of leukemic cells through a signaling pathway alternative to BCR, by over-expression and hyper-activation of LCK and STAT5 hyper-phosphorylation.
Hypothesis on the role of PAX5 fusion proteins to sustain survival of leukemic cells through a signaling pathway alternative to BCR, by over-expression and hyper-activation of LCK and STAT5 hyper-phosphorylation.
