Figure 5.
Longitudinal assessment of renal phenotype in male SCT (AS; n = 18, red lines) and genetic control (AA; n = 18, blue lines) mice. (A) Trajectory of GFR. (B) Urinary protein excretion over time. (C) Urinary osmolality over time. (D) Urinary albumin excretion over time. (E) Urinary KIM-1 excretion over time. Data are mean ± SEM. ∗P < .05; analysis by 2-way analysis of variance with mixed-effect analysis using Bonferroni post hoc test. Panel A, Pinteraction < .0001; Pgenotype = .9004; Page < .0001. Panel B, Pinteraction = .7011; Pgenotype = .0012; Page = .0224. Panel C, Pinteraction = .0469; Pgenotype < 0.0001; Page = 0.0001. Panel D, Pinteraction < .0001; Pgenotype < .0001; Page < .0001. Panel E, Pinteraction = .7150; Pgenotype < .0001; Page = .1897. KIM-1, kidney injury marker 1.

Longitudinal assessment of renal phenotype in male SCT (AS; n = 18, red lines) and genetic control (AA; n = 18, blue lines) mice. (A) Trajectory of GFR. (B) Urinary protein excretion over time. (C) Urinary osmolality over time. (D) Urinary albumin excretion over time. (E) Urinary KIM-1 excretion over time. Data are mean ± SEM. ∗P < .05; analysis by 2-way analysis of variance with mixed-effect analysis using Bonferroni post hoc test. Panel A, Pinteraction < .0001; Pgenotype = .9004; Page < .0001. Panel B, Pinteraction = .7011; Pgenotype = .0012; Page = .0224. Panel C, Pinteraction = .0469; Pgenotype < 0.0001; Page = 0.0001. Panel D, Pinteraction < .0001; Pgenotype < .0001; Page < .0001. Panel E, Pinteraction = .7150; Pgenotype < .0001; Page = .1897. KIM-1, kidney injury marker 1.

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