Model depicting cholesterol emboli, originating from the atheromatous plaques of the abdominal aorta, activate gasdermin D on neutrophils, leading to pyroptosis-mediated neutrophil extracellular trap (NET)osis, thromboinflammation, and subsequent obstruction of microcirculation in the kidney. Figure created using bioRender.

Model depicting cholesterol emboli, originating from the atheromatous plaques of the abdominal aorta, activate gasdermin D on neutrophils, leading to pyroptosis-mediated neutrophil extracellular trap (NET)osis, thromboinflammation, and subsequent obstruction of microcirculation in the kidney. Figure created using bioRender.

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