Figure 4.
WBC count association with TEs at HCT levels ≤45% (WBC count >11 × 109/L and >12 × 109/L). The model included the HR for WBC count (>11 × 109/L vs ≤11 × 109/L and >12 × 109/L vs ≤12 × 109/L) at HCT levels ≤45%; the association between WBC count elevation (>11 × 109/L and >12 × 109/L) and TEs was tested using separate models. Analyses did not identify an association between TEs and WBC count >11 × 109/L; however, a significant association between TEs and a WBC count >12 × 109/L was observed. The blue lines separating rows demark blood count data that were obtained from independent models. The significance for covariates of age, sex, disease duration, history of TE, and treatment with TE occurrence were unchanged across all models; representative data for these covariates from the WBC count >11 × 109/L model are shown (data for all models shown in supplemental Figure 5). Significant values are indicated in blue font.

WBC count association with TEs at HCT levels ≤45% (WBC count >11 × 109/L and >12 × 109/L). The model included the HR for WBC count (>11 × 109/L vs ≤11 × 109/L and >12 × 109/L vs ≤12 × 109/L) at HCT levels ≤45%; the association between WBC count elevation (>11 × 109/L and >12 × 109/L) and TEs was tested using separate models. Analyses did not identify an association between TEs and WBC count >11 × 109/L; however, a significant association between TEs and a WBC count >12 × 109/L was observed. The blue lines separating rows demark blood count data that were obtained from independent models. The significance for covariates of age, sex, disease duration, history of TE, and treatment with TE occurrence were unchanged across all models; representative data for these covariates from the WBC count >11 × 109/L model are shown (data for all models shown in supplemental Figure 5). Significant values are indicated in blue font.

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