Figure 2.
Hb and Hct are inversely correlated with OEF before and after HSCT. Each HSCT recipient’s data points are shown with an arrow oriented from before HSCT to after HSCT. The 95% CI (shaded) and regression line (red) are derived from the OEF and CBF measurements of the CRTT cohort. (A) Correlation between 1/Hb and OEF before HSCT (Spearman's ρ, 0.818; P = .004) and after HSCT (Spearman’s ρ, 0.842; P = .002). (B) Correlation between 1/Hct and OEF before HSCT (Spearman’s ρ, 0.869; P = .0011) and after HSCT (Spearman’s ρ, 0.952; P < .0001). (C) Correlation between 1/Hb and CBF before HSCT (Spearman’s ρ, 0.479; P = .162) and after HSCT (Spearman’s ρ, 0.115; P = .751). (D) Correlation between 1/Hct and CBF before HSCT (P = .162) or after HSCT (P = .726).

Hb and Hct are inversely correlated with OEF before and after HSCT. Each HSCT recipient’s data points are shown with an arrow oriented from before HSCT to after HSCT. The 95% CI (shaded) and regression line (red) are derived from the OEF and CBF measurements of the CRTT cohort. (A) Correlation between 1/Hb and OEF before HSCT (Spearman's ρ, 0.818; P = .004) and after HSCT (Spearman’s ρ, 0.842; P = .002). (B) Correlation between 1/Hct and OEF before HSCT (Spearman’s ρ, 0.869; P = .0011) and after HSCT (Spearman’s ρ, 0.952; P < .0001). (C) Correlation between 1/Hb and CBF before HSCT (Spearman’s ρ, 0.479; P = .162) and after HSCT (Spearman’s ρ, 0.115; P = .751). (D) Correlation between 1/Hct and CBF before HSCT (P = .162) or after HSCT (P = .726).

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