Figure 2.
Posthaploidentical HCT outcomes following the development of CRS stratified by graft source. (A) Cumulative incidence of relapse among patients who did and did not develop CRS, stratified by graft source. Shown is the comparison between CRS and no CRS groups among patients receiving either a BM or PBSC graft. (B) Cumulative incidence of chronic GVHD in patients who did and did not develop CRS and received either a BM or PBSC graft. Shown is the comparison between the CRS and no CRS groups among patients receiving a PBSC graft. (C) Cumulative incidence of moderate-to-severe chronic GVHD in patients who did and did not develop CRS and received either a BM or PBSC graft. Shown is the comparison between the CRS and no CRS groups among patients receiving a PBSC graft. Fourteen of the 81 patients who received a PBSC graft did not develop CRS, and none of these 14 patients developed moderate-to-severe chronic GVHD (dashed blue line on the x-axis).

Posthaploidentical HCT outcomes following the development of CRS stratified by graft source. (A) Cumulative incidence of relapse among patients who did and did not develop CRS, stratified by graft source. Shown is the comparison between CRS and no CRS groups among patients receiving either a BM or PBSC graft. (B) Cumulative incidence of chronic GVHD in patients who did and did not develop CRS and received either a BM or PBSC graft. Shown is the comparison between the CRS and no CRS groups among patients receiving a PBSC graft. (C) Cumulative incidence of moderate-to-severe chronic GVHD in patients who did and did not develop CRS and received either a BM or PBSC graft. Shown is the comparison between the CRS and no CRS groups among patients receiving a PBSC graft. Fourteen of the 81 patients who received a PBSC graft did not develop CRS, and none of these 14 patients developed moderate-to-severe chronic GVHD (dashed blue line on the x-axis).

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