Figure 3.
Impact of CMV reactivation on outcomes according to development of acute GVHD in multivariate analyses. Forest plots show the adjusted HR of CMV reactivation on relapse (A), nonrelapse mortality (B), and OS (C). All models were adjusted for recipient age, sex mismatch, CMV serological status, DRI, HCT-CI, donor source, GVHD prophylaxis, conditioning intensity, in vivo T-cell depletion, year of HCT, and grades 2-4 acute GVHD (G24GVHD) on day 65. In ALL, positivity of Ph-chromosome was also included in the model. Bold indicates statistical significance. ∗P value for the interaction between primary diseases (AML vs ALL) and CMV reactivation. ∗∗P value for the interaction between G24GVHD and CMV reactivation.

Impact of CMV reactivation on outcomes according to development of acute GVHD in multivariate analyses. Forest plots show the adjusted HR of CMV reactivation on relapse (A), nonrelapse mortality (B), and OS (C). All models were adjusted for recipient age, sex mismatch, CMV serological status, DRI, HCT-CI, donor source, GVHD prophylaxis, conditioning intensity, in vivo T-cell depletion, year of HCT, and grades 2-4 acute GVHD (G24GVHD) on day 65. In ALL, positivity of Ph-chromosome was also included in the model. Bold indicates statistical significance. ∗P value for the interaction between primary diseases (AML vs ALL) and CMV reactivation. ∗∗P value for the interaction between G24GVHD and CMV reactivation.

Close Modal

or Create an Account

Close Modal
Close Modal