Figure 3.
C1INH deficiency enhances contact pathway–initiated mouse WB TG. (A) Representative silica-initiated TG curves in WB collected from C1inh+/+, C1inh+/−, and C1inh−/− mice with quantification of TG (B) lag time and (C) peak. (D) Representative TF-initiated TG curves in WB collected from C1inh+/+, C1inh+/−, and C1inh−/− mice with quantification of TG (E) lag time and (F) peak. Data are represented as individual values with median and interquartile range and analyzed by Kruskal-Wallis with post hoc Dunn’s. ∗P < .05.

C1INH deficiency enhances contact pathway–initiated mouse WB TG. (A) Representative silica-initiated TG curves in WB collected from C1inh+/+, C1inh+/−, and C1inh−/− mice with quantification of TG (B) lag time and (C) peak. (D) Representative TF-initiated TG curves in WB collected from C1inh+/+, C1inh+/−, and C1inh−/− mice with quantification of TG (E) lag time and (F) peak. Data are represented as individual values with median and interquartile range and analyzed by Kruskal-Wallis with post hoc Dunn’s. ∗P < .05.

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