Figure 6.
Erythroid-specific MAZ signal is enriched on GATA1-bound enhancers. (A) Genome distribution of MAZ common and erythroid-specific binding sites. (B) Average H3K4me1, H3K4me3, MAZ, and ATAC-seq signal in primary erythroid cells plotted against MAZ erythroid-specific peakset (red) and MAZ common peakset (black) where signal from promoters have been excluded (non-TSS set). (C) Left: Heatmap plot of GATA1 ChIP-seq centered on MAZ erythroid peaks overlapping enhancers (top) and promoters (bottom). Right: Heatmap plot of MAZ ChIP-seq centered on GATA1 peaks overlapping enhancers (top) and promoters (bottom).

Erythroid-specific MAZ signal is enriched on GATA1-bound enhancers. (A) Genome distribution of MAZ common and erythroid-specific binding sites. (B) Average H3K4me1, H3K4me3, MAZ, and ATAC-seq signal in primary erythroid cells plotted against MAZ erythroid-specific peakset (red) and MAZ common peakset (black) where signal from promoters have been excluded (non-TSS set). (C) Left: Heatmap plot of GATA1 ChIP-seq centered on MAZ erythroid peaks overlapping enhancers (top) and promoters (bottom). Right: Heatmap plot of MAZ ChIP-seq centered on GATA1 peaks overlapping enhancers (top) and promoters (bottom).

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